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In tumor tissue, intermittent blood supply leads to an alternating situation of lack-of-oxygen and reoxygenation, which causes oxidative stress

These endocrine abnormalities if not treated, culminate to metabolic syndrome development, in which the key pathophysiological components include abdominal fat accumulation, reduced systemic insulin sensitivity, low-grade inflammation, high blood pressure, high blood sugar levels, high triglyceride levels, and low levels of high-density lipoprotein (HDL) cholesterol [2]

Whether these mutations have a causative or associative role in the ASD phenotype remains to be determined

Pharmacol Res 1995;31:67-72

An analysis of the relative risk for goiter in euthyroid patients with type 2 diabetes