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The cardiovascular risk associated with hyperglycemia in diabetes is explained by the atherogenic process substantiated by the increased ROS formation

*Correspondence: Shunqiong Xiao [email protected] These authors have contributed equally to this work Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

A dose related reduction in body weights of fourth and fifth litter offspring of the treated mating pair occurred during lactation and post-weaning at all doses

phosphorylated GSK3 (Ser9) level was significantly downregulated in Prx II-knockdown HaCaT cells after H 2 O 2 treatment, accompanied by a marked decrease in the phosphorylation levels of its upstream kinase PI3K and AKT (Fig

Therefore, it is logical to objectively assess the consequences of an exercise provocation to understand mechanisms of exertional exhaustion