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The following mechanisms explain a beneficial effect of probiotics through which they might inhibit CIPN: a reduction in proinflammatory cytokine levels, the regulation of the balance between anti-inflammatory and proinflammatory cytokines, the inhibition of neutrophil infiltration and increased intestinal permeability, augmented production of short-chain fatty acids, the inhibition of ammonia formation, changes in the expression levels of toll-like receptors: TLR2 and TLR4, the maintenance of mucosal integrity through mucin secretion and the activation of suppressive T-cell populations [158]

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Importance of bone turnover for therapeutic decisions in patients with CKD-MBD

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Recent studies demonstrate depot-specific adipokine secretion patterns, with visceral fat showing distinct inflammatory profiles compared to subcutaneous deposits