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Maintenance of redox homeostasis typically involves the delicate regulation of ROS by redox systems, with evidence that the glutathione (GSH/GSSG), thioredoxin and cysteine/cystine redox systems are differentially modulated under dynamic and non-equilibrium redox conditions [16]

299 Three NPC subtypes (immunogenic, classical, and mesenchymal) have been identified as molecularly distinct and clinically relevant, of which the mesenchymal subtype (~ 36%) is associated with poor prognosis, characterized by suppressing tumor suppressor miRNAs and the activation of EMT

Crosstalk between metabolism and circadian clocks

Unlike classical mediators such as TNF-, the activity of RvE1 is tightly confined within an immune-moderate response range, preventing excessive inflammatory damage

CD81 (cat