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Structural matrix repair: GHK-Cu drives Type I collagen synthesis in intestinal fibroblasts, supporting the submucosal connective tissue layer

This suggests that excessive GHK-Cu (or other copper peptides) might theoretically push skin MMP-1 concentrations beyond levels supporting healthy collagen production into ranges associated with collagen fragmentation and breakdown seen in older skin with naturally elevated MMP-1

Potential of TB-500 & BPC-157 Blend on Ligaments and Tendon Fibroblasts Laboratory studies by Xu et al

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The researchers commented that the peptide suppressed caspase-dependent apoptosis that increases during aging of cell cultures