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Mitochondrial metabolic reprogramming in CRC: mechanisms of therapy resistance and emerging interventions As discussed above, CRC cells adapt to hypoxia, nutrient deprivation, and therapeutic stress through mitochondrial metabolic reprogramming, which maintains redox homeostasis and organelle quality control to promote survival

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Among those affected, 87.9% reported that their symptoms interfered with work, home, or social functioning

Finally, neither the ShHTL1ShMAX2 (Fig