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Description

For many men, these changes are linked to one key factor: low testosterone

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and continuous pro-synthetic signaling can tip toward fibrosis and scarring rather than functional repair

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Franco R et al

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References 1.Council for Agricultural Science and Technology

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(70) utilized a network pharmacological approach to predict anti-renal tubulointerstitial fibrosis effects via the ERK1/2 signaling pathway, a finding later confirmed in human renal tubular epithelial cells induced by high glucose

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