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Because less of the peptide is likely to be degraded in the stomach during oral studies, researchers may find that different dosing models are required to achieve the same systemic exposure compared to the acetate form

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TLR3 activation augments matrix metalloproteinase production through reactive nitrogen species generation in human lung fibroblasts

Disease models that recapitulate pathophysiological mechanisms and phenotypes of mitochondrial and neurodevelopmental disorders remain a fundamental bottleneck in order to identify new compounds and successfully translate them to clinical practice