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The new study points toward more precise strategies: Blocking downstream mediators such as osteopontin Reprogramming macrophages toward a pro-inflammatory phenotype Combining TGF- pathway inhibition with checkpoint blockade Using localized or selective TGF- suppression Engineering T cells resistant to TGF- signaling In preclinical models, blockade of TGF- restored T-cell infiltration and enabled formerly resistant tumors to respond robustly to immunotherapy