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In vivo endothelial cell-specific knockout of MET (through the generation of Tie-Cre Met fl/fl mice) led to normalised vasculature, reduced intra-tumoural hypoxia, slowed glioblastoma growth and importantly, sensitised glioblastoma tumours to temozolomide treatment, prolonging mouse survival [40]

The dihexa half life only matters if the compound reaching circulation is structurally intact

The peptide's gastric stability allows oral effectiveness, particularly for digestive issues

The timing of MOR activation can also determine whether it can enhance CA1 function

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