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Such a defect can lead to a dangerously small pool of RG, reducing the number and diversity of future neurons and glia (Subramanian et al., 2020)

Notably, supplementation with ferrostatin-1 or trolox, both of which are radical-trapping antioxidants and potent inhibitors of ferroptosis, rescued survival but not proliferation under cystine-free conditions (Extended Data Fig

However, in Lewy body disease with comorbid AD, Lewy-related pathology in the amygdala is found from an early stage and even in the absence of Lewy-related pathology in the brainstem [192, 193, 334]

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