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We found that the number of TUNEL-positive cells increased in the ACLT group, while l-carnitine inhibited this upregulation (Fig

10.1016/j.neulet.2016.02.011 103 DeGregorio-RocasolanoN.Mart-SistacO.GasullT

Although endothelial activation and redox signaling compose a normal host defense, when this activation is sustained, chronic production of ROS may exceed the capacity of cellular enzymatic and nonenzymatic antioxidant defenses [49], contributing to triggering vascular diseases such as atherosclerosis or peripheral artery disease [102, 103]

Collectively, these interconnected mechanisms highlight that drought tolerance emerges from dynamic coordination among redox signaling, hormonal regulation, and metabolic plasticity rather than from single-pathway responses

10.2147/COPD.S314349 85 LibiadM.VitvitskyV.BostelaarT.BakD