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For example, after knockout of -arrestin 2, mGluR1/5-mediated plasticity of CA3 and CA1 pyramidal neurons was impaired, suggesting that there is a non-classical signaling pathway dependent on -arrestin 2 in mGluR1 [34]

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Inhibition of upstream platelet-derived growth factor receptor has been found to reduce TGF- expression, offering a promising anti-fibrotic pathway [209]