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In cardiovascular diseases other than hypertension, endothelial dysfunction is strongly and independently associated with cardiovascular events

Overexpression of NLGN3 or 3A2 increases evoked inhibitory postsynaptic currents (IPSCs), while overexpression of NLGN3A1 or 3A1A2 leads to a reduction in IPSC amplitude

haematobium seemingly have similar invasion kinetics to what is seen in rodent model systems, with penetration into the dermis and location of a venule taking between 48 and 72 h (7678)

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This approach revealed that the metabolic compensatory pathways that enable cells to overcome glutamine depletion are contingent on the capacity to synthesize glutamine via glutamine synthetase