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10.3389/fnins.2019.00384 22

doi:10.1093/ecam/nem040 References are provided for educational context

Although Cyb5r3 -KD MLE12 cells did not show any pro-fibrotic phenotype on baseline (data not shown), they exhibited a higher expression of the senescence marker Cdkn1a , which was highly increased upon TGF-1 treatment and not induced in the Cyb5r3 -Scr cells (Figure 6B)

This protective ability is due to its ability to decrease oxidative stress, modulate inflammatory cytokines, and inhibit apoptosis, as shown in studies in other non-central inflammatory conditions [5, 24,25,26]