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Table S6 )

The TH production, per se , is a high source of ROS in the thyrocyte

Obesity and insulin resistance induce early development of diastolic dysfunction in young female mice fed a western diet

These compounds induce excessive ROS production by depleting antioxidants, activating enzymatic ROS-generating systems (e.g., NADPH oxidase), and triggering mitochondrial dysfunction, ultimately impairing spermatogenesis and sperm quality ( Figure 1g ) ( Despite their diverse origins, both endogenous and exogenous factors converge on a common pathogenic pathway: by inducing mitochondrial dysfunction, activating enzymatic ROS-producing systems, or impairing antioxidant defenses, they lead to excessive ROS accumulation

Still, you must note that specific side effects can vary based on the individual, the type of antioxidants used, and other factors