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A recent editorial underscores that ferroptosis, driven by oxidative stress, mitochondrial dysfunction, and immune dysregulation, is a key mechanistic node in PD and Alzheimers disease (AD), highlighting its potential as both a biomarker and a therapeutic target (Dar et al., 2024)

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Research literature frequently references both peptides individually in experimental studies involving peptide chemistry, molecular biology, biochemical analysis, and preclinical laboratory investigations

Occupational exposure to heavy metals: DNA damage induction and DNA repair inhibition prove coexposures to cadmium, cobalt and lead as more dangerous than hitherto expected

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