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These findings suggest that impaired methylation reactions and/or protein synthesis may not be the main mechanisms underlying moderate hyperhomocysteinemia-induced vascular disease

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Here we identify an underappreciated role for GSH as a cysteine reservoir for tumours

2114.10.1038/s41598-023-29305-5 Hao J et al (2023) Combination treatment with FAAH inhibitors/URB597 and ferroptosis inducers significantly decreases the growth and metastasis of renal cell carcinoma cells via the PI3K-AKT signaling pathway Cell Death Dis, 14(4): p