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Significantly reduced GPX4 levels in MCAO/R mouse or rat brains and in oxygen-glucose deprivation/reperfusion (OGD/R)-treated hippocampal neurons have the potential to exacerbate lipid peroxidation and ferroptosis

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Known as the bodys "master antioxidant," it plays a central role in protecting cells and supporting detoxification

Local alignment of ligand binding sites in proteins for polypharmacology and drug repositioning

While low dose endotoxin triggered NAMPT-dependent NAD + salvage pathway to support pro-inflammatory response, high dose endotoxin drove a switch from NAD + salvage to IDO1-dependent NAD + de novo biosynthesis at late stage, leading to persistent anti-inflammatory properties and immune tolerance [98]