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By upregulating its expression, it can promote inhibitory signaling (87)

Cells were incubated for 30 min in BFP media containing 5 M BODIPY-C11

Next, we investigated whether this system could be used for multiple deletions

doi: 10.1038/mp.2013.7

This indicates that, despite the high correlation of promoter methylation with ACB expression and drug sensitivity, epigenetic silencing is not a primary cause for the lack of ACB induction by NRF2 and the inability of SCLC to adapt to drug-induced ROS stress