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However, the study showed that BDNF-TrkB signaling plays a necessary role in mediating CSDS-induced social avoidance behavior in the ventral tegmental area (VTA)-NAc, and established BDNF-TrkB signaling as a pathological mechanism during chronic stress [116]

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However, their dysfunction, marked by impaired oxidative phosphorylation, excessive oxygen species (ROS) production, and defective quality control, disrupts these processes and causes cellular and organ damage

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