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The 2025 mechanistic work reporting AMY1R and AMY3R dependence is a good example of studies that aim to explain why the compound performs as it does at the receptor system level

Their cost-effectiveness and favorable pharmacokinetic properties (e.g., efficient absorption and potent bioactivity) make them attractive candidates for localized and targeted wound therapies [29, 54-57]

Activation of Tissue Remodeling Proteases GHK-Cu activates matrix metalloproteinases (MMPs) and their inhibitors (TIMPs) in a balanced way that facilitates controlled extracellular matrix remodeling breaking down damaged collagen and replacing it with new, organized fibrillar collagen rather than scar tissue

Even a recent 2026 review concludes the same thing that GHK-Cu appears safe but the evidence is largely preclinical and comprehensive human RCTs are lacking (Wojcieszuk et al., 2026)

All investigations are performed in laboratory settings only