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These observations led us to hypothesize that inflammatory skin-imprinted eosinophils may limit T cell function by competing with them for glucose, particularly affecting the highly glycolytic Th1 cells (Fig

FowlerBrown A, Kim DH, Shi L, et al

Apoptosis-stimulating protein two of p53 (ASPP2), the binding partner of p53, has been found to suppress liver regeneration by inhibiting the mTORC1 pathway