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Musculoskelet Surg

Tree Physiol 43:10091022

Conclusion The past two decades have witnessed a paradigm shift in the study of MuSC aging, fueled by continuous advances in transcriptomic technologies and the analytical frameworks built around them

It is still unclear whether mHTT initiates ferroptosis by suppressing ferroportin or activating NCOA4-mediated ferritinophagy to disrupt striatal iron homeostasis, nor is it known how lipid peroxidation products generated during ferroptosis (such as 4-HNE) modify mHTT to accelerate its nuclear aggregation and toxic propagation