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Hirose T, Shimizu K, Ogura H, Tasaki O, Hamasaki T, Yamano S, et al

As a result, hepatocellular supplies of glutathione become exhausted and NAPQI is free to react with cellular membrane molecules, resulting in widespread hepatocyte damage and death, leading to acute hepatic necrosis

The ultrastructure of the liver tissue was examined using transmission electron microscopy

There are a number of problems with this however

5.4 The nuclear factor erythroid 2-related factor 2 (Nrf2) The Nrf2 pathway serves as a critical endogenous defense mechanism against OS in HTN, orchestrating the transcription of antioxidant and cytoprotective genes that counteract vascular dysfunction (Lal et al., 2024)