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In many cell types, RVI is known to be produced by shrinkage-induced NaCl uptake through activation of NHE, NKCC, and/or HICC (Okada et al., 2019)

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J Nucl Med 55:945950 Nguyen T, Johnston S, Clarke L et al (2017) Impaired calcium mobilization in natural killer cells from chronic fatigue syndrome/myalgic encephalomyelitis patients is associated with transient receptor potential melastatin 3 ion channels