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Acknowledgements This research was supported in part by AMED-CREST, by JST, PRESTO, by MEXT/JSPS KAKENHI grant numbers JP16H02606 and JP26111012 (to Y.U.), JP15H05951 Resonance Bio (to M.K.), by JSPS Core-to-Core Program, by a grant from Hoansha Foundation (to Y.U.)

Programmed aging of mammals: Proof of concept and prospects of biochemical approaches for anti-aging therapy

SIRT2 directly interacts with GCLC As SIRT2 could desuccinylate GCLC, we then explored the physical association between SIRT2 and GCLC, especially under oxidative stress condition

Excess ROS can damage cellular biomolecules, including proteins, DNA, and lipids, leading to fibroblast degeneration [23]

In conclusion, we found that dietary glycine supplements corrected obesity-associated glycine deficiency, increased urine acylglycine excretion, and plasma metabolites associated with one-carbon metabolism