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The compound appears to work by increasing intracellular NAD+ levels (through preventing nicotinamide consumption by NNMT), which activates sirtuins and enhances mitochondrial function and fatty acid oxidation, while also promoting a phenomenon called "browning" of white adipose tissue - the transformation of energy-storing white fat cells into metabolically active beige/brown adipocytes that burn fatty acids for thermogenesis rather than storing them

Compound Overview AHK-Cu is a synthetic copper peptide complex studied in biochemical research for its interaction with signalling pathways and molecular regulatory systems

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I am hoping there is no permanent long term damage to my heart and lungs