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Iron chelators like deferoxamine and deferiprone reduce labile iron and mitigate reactive oxygen species (ROS), yet they face limitations in crossing the BBB effectively

doi: 10.3389/fmed.2021.645816 153 KapsogeorgouEKAbu-HeluRFMoutsopoulosHMManoussakisMN

Many treatment strategies aiming to directly disrupt the formation of amyloid (A) plaques and neurofibrillary tangles (NFTs), the two major AD hallmarks, showed modest or no benefit [2]

Its key step involves kinase cascade-mediated activation and oligomerization of MLKL, which subsequently disrupts plasma membrane integrity (102)

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