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Description

Najar 2016

demonstrated that ZDHHC5-mediated palmitoylation of beclin 1 serves as a core mechanism for maintaining neuronal autophagy, and its deficiency in AD murine models impairs autophagic function, exacerbating proteotoxic deposition, thereby leading to neurodegeneration, long-term potentiation (LTP) deficits, and memory deficits (167)

Quercetin enhances 5-fluorouracil sensitivity by regulating the autophagic flux and inducing drp-1 mediated mitochondrial fragmentation in colorectal cancer cells