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A randomized, double-blind phase I/IIa study of intranasal glutathione in Parkinsons disease

Oxidative stress exaggerates disease condition by interfering with various signaling pathways like RCAN1, CREB/ERK, Nrf2, PP2A, NFB and PI3K/Akt

Neurological disorders, both acute and chronic also signify low glutathione

In contrast, vascular endothelial cells, a major target of hyperglycemic damage, show no significant change in glucose transport rate when glucose concentration is elevated, resulting in intracellular hyperglycemia (Figure 35.1)

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