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Makino SI, Shirata N, Oliva Trejo JA, Yamamoto-Nonaka K, Yamada H, Miyake T

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Its levels naturally decline with age, correlating with slower wound healing and loss of skin elasticity

The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes