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This is likely induced through interactions with toll-like receptor 4 (TLR4), a key receptor in proinflammatory downstream cellular signaling including the release of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-)

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Results: In the RADIcAL2 cohort, 38% of patients had no rejection, while 62% had either indeterminate (21%) or T-cell mediated rejection (41%)