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These involve normal epithelial and fibroblast cells via upregulation of telomerase

In terms of molecular mechanisms, ZDHHC3 loss of function resulted in reduced palmitoylation modification of inactive rhodopsin 2 (IRHOM2), which in turn promoted its degradation via the ubiquitin-proteasome pathway and ultimately inhibited the activation of the downstream MAP3K7-JNK signaling pathway (158)

Data availability The data used to support the findings of this study have not been made available because they are the property of the Pomeranian Medical University in Szczecin