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These data represent an in vivo demonstration of the importance of the modulation of oxidative stress as part of the anti-neoplastic activity of UT, as well as constitute evidence of the lack of activity of isolated POAs in the primary tumour of this tumour lineage

10.1124/dmd.107.01490217496207 69 Caulfield MJ, Munroe PB, ONeill D, et al

What's the basic mechanism difference

Like CD8 + T cells, CD4 + T cell activation depends on glycolysis, glut1 deficiency reduces thymocyte counts, suppresses the expansion of effector CD4 + T cells, and impairs cytokine production in various CD4 + T cell subsets, including interferon-, IL-4, and IL-17 (238)

This is evident by the strong odor of hydrogen sulfide