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lips-14 c elegans

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. The Heterochronic Gene lin-14 Controls

The Heterochronic Gene lin 14 Controls Axonal Degeneration in C. elegans Neurons ScienceDirect Lipid Oxidation at the Crossroads: Oxidative Stress and Neurodegeneration Explored in Caenorhabditis elegans Inactivation of SIAH 1 E3 ligase attenuates A toxicity by suppressing ubiquitin dependent DVE 1 degradation in Caenorhabditis elegans models of Alzheimer's disease Journal of Biological Chemistry PGP 14 establishes a polar lipid permeability barrier within the C. elegans pharyngeal cuticle PLOS Genetics Handbook Nervous System Neuronal Support Cells

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lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. The Heterochronic Gene lin-14 Controls

Figure 6AH show a marked accumulation of Rab7 and Arl8 in the axons and dendrites of old age nTg neurons (Fig

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. The Heterochronic Gene lin-14 Controls

Empirical formula C Molar mass (M) 349,36 g/mol Storage temp

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. The Heterochronic Gene lin-14 Controls

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lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. The Heterochronic Gene lin-14 Controls

K.PathakC

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. The Heterochronic Gene lin-14 Controls
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