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Elevated levels of pro-inflammatory cytokines, such as Interleukin-6 (IL-6) and Tumor Necrosis Factor-alpha (TNF-), generate massive amounts of reactive oxygen species (ROS)

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This synergy effect may arise because pathway inhibitors can block the downstream survival signals, while DNA methyltransferase inhibitors (DNMTi) can restore the expression of tumor suppressor genes, making cells more sensitive to apoptosis