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In particular, during ferroptosis, AECs collapse due to iron overload and lipid peroxidation-induced damage, releasing large amounts of ROS, which further activate fibroblasts and enhance ECM deposition, thereby accelerating pulmonary tissue stiffening (Cheng et al., 2021a)

Posttranslational stability of the heme biosynthetic enzyme ferrochelatase is dependent on iron availability and intact iron-sulfur cluster assembly machinery

The reversal of NET function results in efflux of cytoplasmic norepinephrine, normally contained in the , readily releasable pool 12) into the synaptic cleft, independent of calcium influx and the normal process of vesicular release (Raiteri and Levi, 1986

Both refer to the same treatment

rs641738T SNP favors hepatic fat accumulation and contributes to various liver diseases, including NAFLD, fibrosis, and HCC