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This selectivity stems from the unique dependence of senescent cells on the FOXO4-p53 interaction for survival, a vulnerability not present in proliferating or quiescent healthy cells

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However, when it is absent, as in Piezo1 KO embryos, the level of cholesterol synthesis in the CNS and the levels of neutral lipids and cholesterol in NSCs are reduced (Nourse et al., 2022)