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Pseudomonas normally thrives in the small bowel and produces abundant cobalamin that may be a significant source for the human host.[6] P

doi: 10.1080/10715760802582183

Combined treatment with MPE and LGG significantly restored colon morphology, reduced spleen hypertrophy, and suppressed inflammatory cytokines and enhanced GLP-1, Nrf2 expression and SCFA levels, indicating enhanced gut barrier function and microbial homeostasis

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Conversely, iron chelators reverse the glucose-induced LDH release and alterations in ferroptosis-related genes in mouse mesangial cells, indicating that high glucose can induce ferroptosis in mesangial cells (124)