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In addition to lipid accumulation and inflammation, this variant influences the development and progression of MASLD by stimulating oxidative stress

Protein chemical synthesis combined with mirror-image phage display yields D-peptide EGF ligands that block the EGF-EGFR interaction

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Immunohistochemistry of liver sections showed CD36 as located in the hepatocyte plasma membrane in patients with steatosis and NASH, compared to a weak CD36 expression confined to the cytoplasm of hepatocytes in normal livers, suggesting that the translocation of CD36 protein from cytoplasm to membrane may be a triggering event in NAFLD progression [25]