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RS is an autosomal dominant disorder in which all affected individuals reported to date are heterozygous at the H1-4 locus and therefore express both mutant and WT H1.4

This underscores its potential as a supportive treatment and addresses the interplay between metabolic and inflammatory aspects of the disease

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If regulated compounding expands access to certain peptides, clinics may evaluate how these therapies fit within existing longevity, metabolic health, or performance medicine offerings