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[4] The authors themselves concluded the effect appeared in a small number of subjects and that long-term safety was not established

Endoplasmic reticulum stress/Ca(2+)-calmodulin-dependent protein kinase/signal transducer and activator of transcription 3 pathway plays a role in the regulation of cellular zinc deficiency in myocardial ischemia/reperfusion injury

The second mechanism requires scavenging protein hemopexin (Hx)