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It is worth noting that the primary strategy to induce ferroptosis currently involves either genetic knockout or pharmacological inhibition of GPX4 [123]

In addition, senescent cells can also activate the DDR in neighbouring cells via gap junction-mediated cell-cell contact in a process that involves oxidants, resulting in the induction of what is known as secondary senescence (Nelson et al., 2012)

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Moreover, stress-triggered upregulation of nuclear factor erythroid 2-related factor 2 (NRF2) and ATF4 in DTPs inhibits lineage-specific chromatin accessibility and activates dedifferentiation or EMT programs, thereby improving cell survival during treatment [33]

Both peptides are produced to 99% purity (HPLC tested) and provided in lyophilized powder form to ensure stability and accuracy in research use