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A recent study reported that activation of AMPK1 promoted PASMCs proliferation [23]

assessing a panel of six stimuli showed that monocyte-derived macrophages from individuals with CD were more resistant to apoptosis induced by phorbol 12-myristate 13-acetate (PMA) or TNF compared to healthy controls: they suggested that the prolonged presence of these macrophages would contribute to inflammation and limit tissue repair [9]

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