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Antagonism of RANTES receptors reduces atherosclerotic plaque formation in mice

As part of these efforts, cNF research should leverage the power of current technologies developed for oncology research

its optimal, high intracellular concentration, is of the utmost importance and can be increased by administration of reduced glutathione or - in the case of a disturbed cell metabolism - can in any case be restored

Besides this activation mechanism it is also known that PKG can be activated under oxidative stress independent of the respective cGMP or NO levels [172]