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Acknowledgements The authors wish to acknowledge grant support from the German Research Foundation (DFG), Clinical Research Unit KFO 181, projects 1 (STE 892/9-2) and 6 (SCHA 1531/1-1)

These mutations disrupt iron homeostasis by phosphorylating Rab8a, leading to the sequestration of Tf within lysosomes and potentially promoting iron accumulation and ferroptosis