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Activated glial cells serve a crucial role in neuroinflammation and release soluble signaling molecules, including chemokines, pro-inflammatory cytokines, glutamate, prostaglandins, ROS, RNS and damaged mitochondria (62-64)

Clinical translation requires addressing challenges including cell-type specificity, development of disease-specific ferroptosis biomarkers, optimization of combination regimens, and careful safety assessment to avoid unintended effects such as myelosuppression or hepatorenal toxicity

XW, MI, SA, FS, MM, MH-O-R: Collection of data and analysis

Participants were excluded from the study if they suffered from the following diseases: alcohol use disorder, diabetes mellitus, kidney or bladder stones, kidney disease, liver disease, or stomach ulcers

10.1038/mp.2017.249 33