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At the regulatory level, aberrant activation of canonical pro-inflammatory pathways such as NF-B disrupts junctional complexes and induces cytokines, including IL-1, thereby amplifying inflammation and further compromising barrier integritya self-perpetuating cycle (Zeng et al., 2024)

In vivo reprogramming reduces systemic vitamin B 12 levels Our whole-genome approach allowed us to investigate changes not only in bacterial species abundance, but also in gene composition and ontology groups, which could uncover pathways relevant to reprogramming

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