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However, translating these promising findings into clinical success remains a formidable challenge

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The subsequent phase of microbial metabolite dysregulation rectification involves polysaccharide-mediated facilitation of SCFA biosynthetic machineryparticularly optimizing propionate, acetate, and isobutyrate metabolic fluxwhile punicalagin concurrently executes precision targeting of circulating endotoxin neutralization and diamine oxidase level modulation

Intriguingly, members belonging to the same subfamily exhibit similar cellular locations and functional characteristics 1 (Fig